Animal Models in Cardiovascular Research by David R. Gross D.V.M., Ph.D. (auth.)

By David R. Gross D.V.M., Ph.D. (auth.)

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0 Conscious, previously instrumented, intact Used as a preanesthetic. Onset of action within a few minutes following injection. 1: Initial transient decrease in coronary resistance, initial increase in heart rate and in left ventricular dP/dt and dP/dt/P, decrease in left ventricular end-diastolic diameter and left ventricular end-systolic size. These responses followed by an increase in coronary resistance lasting 5-30 min. The heart rate, aortic pressure and left ventricular end-diastolic diameter returned to baseline values but left ventricular dP/dt/P remained elevated.

0 Conscious, previously instrumented, intact Used as a preanesthetic. Onset of action within a few minutes following injection. 1: Initial transient decrease in coronary resistance, initial increase in heart rate and in left ventricular dP/dt and dP/dt/P, decrease in left ventricular end-diastolic diameter and left ventricular end-systolic size. These responses followed by an increase in coronary resistance lasting 5-30 min. The heart rate, aortic pressure and left ventricular end-diastolic diameter returned to baseline values but left ventricular dP/dt/P remained elevated.

No change in pulmonary, aortic or right atrial pressures. No change in blood gases or respiratory rate 2 2 53 Onset in about 10 min, with duration of about 1 hour. 1 min No data Rate reduced, decreased contractile amplitude. In the electrically driven atria there was an initial positive inotropic response which preceeded the negative inotropic reaction. When the calcium ion concentration of the preparation was increased the Pentazocine induced decrease in contractility was antagonized. 03 mg/kg Conscious, previously instrumented.

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