High-Risk Atherosclerotic Plaques Mechanisms, Imaging, by Levon Michael Khachigian

By Levon Michael Khachigian

Weak plaque improvement is the results of a posh sequence of molecular and mobile occasions concerning irritation, apoptosis, rupture, and thrombosis. a close realizing of the mechanisms underlying the improvement of high-risk plaques, besides the facility to imagine and diagnose those weak lesions, will bring about the powerful administration of acute coronary syndromes.
High-Risk Atherosclerotic Plaques: Mechanisms, Imaging, types, and treatment brings jointly well timed, in-depth reports via well known foreign cardiologists and scientists. Chapters hide the definition, constitution, and mobile and molecular mechanisms of excessive threat plaque improvement, in addition to animal versions of susceptible plaque, plaque imaging, and present and destiny cures. medical examiners speak about intravascular ultrasound, optimum coherence tomography, magnetic resonance imaging, and coronary thermography. the ultimate bankruptcy stories either present and destiny neighborhood and systematic options for the healing administration of weak plaque.
Exploring all features of this first explanation for acute coronary syndromes, this informative e-book updates our wisdom at the detection and therapy of susceptible plaques. it's a necessary source which can drastically increase the development in therapy and prevention.

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35. Kockx MM. Apoptosis in the atherosclerotic plaque: quantitative and qualitative aspects. Arterioscler Thromb Vasc Biol 1998, 18: 1519–1522. 36. Boyle J, Bowyer D, Weissberg P, and Bennett M. Human blood-derived macrophages induce apoptosis in human plaque-derived vascular smooth muscle cells by Fas ligand/Fas interactions. Art Thromb Vasc Biol 2001, 21: 1402–1407. 37. Boyle JJ, Weissberg PL, and Bennett MR. Human macrophage-induced vascular smooth muscle cell apoptosis requires NO enhancement of Fas/Fas-L interactions.

6. Bauriedel G, Hutter R, Welsch U, Bach R, Sievert H, and Luderitz B. Role of smooth muscle cell death in advanced coronary primary lesions: implications for plaque instability. Cardiovasc Res 1999, 41: 480–488. 7. Mallat Z, Hugel B, Ohan J, Leseche G, Freyssinet JM, and Tedgui A. Shed membrane microparticles with procoagulant potential in human atherosclerotic plaques: a role for apoptosis in plaque thrombogenicity. Circulation 1999, 99: 348–353. 8. Mallat Z, Benamer H, Hugel B, Benessiano P, Steg P, Freyssinet J, and Tedgui A.

Niemann-Jonsson A, Ares MP, Yan ZQ, Bu DX, Fredrikson GN, Branen L, PornAres I, Nilsson AH, and Nilsson J. Increased rate of apoptosis in intimal arterial smooth muscle cells through endogenous activation of TNF receptors. Arterioscler Thromb Vasc Biol 2001, 21: 1909–1914. 50. Boyle J, Weissberg P, and Bennett M. Tumour necrosis factor promotes macrophageinduced vascular smooth muscle cell apoptosis by direct and autocrine mechanisms. Arterioscl Thromb Vasc Biol 2003, 23: 1553–1558. 51. Seshiah PN, Kereiakes DJ, Vasudevan SS, Lopes N, Su BY, Flavahan NA, and Goldschmidt-Clermont PJ.

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