Sudden Cardiac Death: A Handbook for Clinical Practice by Silvia Priori, Douglas Zipes

By Silvia Priori, Douglas Zipes

This booklet attracts at the tested eu instructions from the ESC that tackle the most important matters in unexpected cardiac loss of life, similar to selecting members in danger sooner than an episode of a ventricular tachyarrhythmia or a surprising cardiac arrest, and responding swiftly to the individual soreness the development out-of-the-hospital. It offers an replace on what's identified approximately unexpected cardiac arrest, from simple experimental reports to medical trials, and serves as a supplement to the ESC center Syllabus in this subject.

Topics contain epidemiology, genetics, arrhythmogenic mechanisms, probability stratification, autonomic fearful procedure and phenotypes. ailment states and specific populations also are lined, in addition to drug, machine and ablation remedies, and price effectiveness. All chapters are co-authored through specialists from either Europe and the US.

The ESC schooling Series
This booklet is a part of the ESC schooling sequence. The sequence is designed to supply doctors with the newest information regarding the certainty, analysis and administration of cardiovascular illnesses. the place on hand, administration techniques are according to the confirmed ecu instructions, which surround the easiest recommendations to exploit with each one cardiac sickness. during the sequence, the prime foreign opinion leaders were selected to edit and give a contribution to the books. the knowledge is gifted in a succinct and available structure with a medical focus.

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Extra resources for Sudden Cardiac Death: A Handbook for Clinical Practice (European Society of Cardiology)

Sample text

In adolescent and young adult populations, the risk is about 1% of that of the general adult population, and familial diseases, such as the congenital long QT syndrome, hypertrophic 33 Silvia: “chap03” — 2005/10/6 — 22:31 — page 33 — #1 34 Chapter 3 cardiomyopathy, arrhythmogenic right ventricular dysplasia, and Brugada syndrome play a dominant role [6]. Mechanisms of ventricular fibrillation McWilliam [7] was the first to suggest that disturbances in impulse propagation could be responsible for fibrillation, and he clearly envisaged the possibility that myocardial fibers could be reexcited as soon as their refractory period had ended.

The salvos of triggered activity in in vitro preparations are slower than the tachycardias in vivo. It is likely that the delayed afterdepolarizations, and the triggered activity, might initiate reentrant rhythms that lead to ventricular fibrillation. The electrophysiological changes associated with heart failure have been the focus of intense research [65]. Heterogeneous action potential prolongation results as a function of myocardial remodeling [66], leading to an increase in the electrophysiological heterogeneity found in the normal ventricle [67].

Two periods of early ventricular arrhythmias in the canine acute myocardial infarction model. Circulation 1979; 60: 397–403. 34. Smith WT, Fleet WF, Johnson TA et al. The 1B phase of ventricular arrhythmias in ischemic in situ porcine heart is related to changes in cell-to-cell coupling. Circulation 1995; 92: 3051–3060. 35. Cinca J, Warren M, Carreno A et al. Changes in myocardial electrical impedance induced by coronary artery occlusion in pigs with and without preconditioning: correlation with local ST-segment potential and ventricular arrhythmias.

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